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Fig. 1 | BMC Endocrine Disorders

Fig. 1

From: Investigation of the association between habitual dietary FODMAP intake, metabolic parameters, glycemic status, and anthropometric features among apparently healthy overweight and obese individuals

Fig. 1

Graphical abstract of consuming different amounts of FODMAPs on cardiometabolic factors. Consumption of low and medium amounts of FODAMPs can work through the effect on the microbial population and their metabolites in lipid and glucose metabolism, as well as adipogenic differentiation. While high FODMAPs in the diet due to the high amount of fructose with the effect of increasing the absorption of sodium and chloride and reducing their excretion causes an increase in blood pressure, also when the liver is exposed to high fructose, the accumulation of triglycerides causes a decrease in insulin sensitivity and then insulin resistance. FODMAP, fermentable oligosaccharides, disaccharides, monosaccharides and polyols; SCFA, Short-chain fatty acids; PPAR-γ, peroxisome proliferator-activated receptor-γ, PYY, peptide YY; GLP-1, glucagon-like peptide 1; CCL, cholecystokinin; TG, triglyceride, IR, insulin resistance

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BMC Endocrine Disorders

ISSN: 1472-6823

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