Skip to main content
Fig. 5 | BMC Endocrine Disorders

Fig. 5

From: Abnormal phosphorylation of Tie2/Akt/eNOS signaling pathway and decreased number or function of circulating endothelial progenitor cells in prehypertensive premenopausal women with diabetes mellitus

Fig. 5

The Tie2/Akt/eNOS signaling pathway of circulating EPCs in the three groups. There was no difference in the phosphorylation of tie2 a, Akt b and eNOS c of circulating EPCs between prehypertensive and normotensive premenopausal women without diabetes mellitus. The phosphorylation of tie2 a, Akt b and eNOS c of circulating EPCs in prehypertensive and normotensive premenopausal women without diabetes mellitus exceeded that in prehypertensive premenopausal women with diabetes. The expression of tie2 a, Akt b and eNOS c of circulating EPCs in the three groups exhibited no difference. Data are given as mean ± SD. (*P < 0.05 compared with normotension without diabetes mellitus, # P < 0.05 compared with prehypertension without diabetes mellitus, n = 20 for each group)

Back to article page